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For example, cyst-lining epithelial cells (the primary drivers of ADPKD progression) may exhibit distinct ferroptosis vulnerabilities compared to adjacent normal tubular epithelial cells, yet the molecular basis for this difference (e.g., unique lipid metabolic profiles and antioxidant capacity) has not been rigorously characterized

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In the CNS, zinc released from nerve endings activates the MAPK pathway, inducing neuronal apoptosis, MMP-9-dependent BBB disruption, and microglial activation, which in turn releases proinflammatory cytokines, ultimately damaging the myelin sheath [284]

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Indeed, while examining dimerization between the D2 receptor and GHSR1a, Kern et al