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In islet cells, the brain, and renal tubular cells, the reaction is directed primarily towards the oxidative deamination of glutamate because these cells and tissues contain high glutamate and low -KG/NH3 levels ( FIGURE 2 GDH aids in preserving the NH4 + concentration in the kidneys and several other organs ( + levels and low glucose levels ( Mutations in short-chain 3-hydroxyacyl-CoA dehydrogenase (SCHAD), which is involved in the oxidation of fatty acids, can also result in hyperinsulaemia and hypoglycaemia ( Research on ADP-ribosylation suggested that this alteration also plays a part in mediating the effect of GDH on insulin synthesis

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