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Besides stimulating POMC transcription and ACTH biogenesis, CRH stimulates the release of ACTH from corticortophs via CRH-R1 leading to a biphasic response with the fast release of a pre-synthesized pool of ACTH, and the slower and sustained release of newly-synthesized ACTH (57)
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Acute respiratory distress syndrome (ARDS) involves neutrophil accumulation in alveolar spaces and NSP release, causing alveolar-capillary barrier damage and exacerbated inflammatory responses (Donnelly et al., 1995)
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