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GregoryJCBuffaJAOrgEWangZLevisonBSZhuWet al

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Concurrently, increased aconitate serves as a substrate for aconitate decarboxylase 1 (ACOD1), producing itaconatean immunoregulatory metabolite that inhibits succinate dehydrogenase, stabilizes Nrf2 via KEAP1 alkylation, and suppresses excessive IL-1 and type I interferon responses and constrain inflammation 103,104