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These effects are real but work at a systemic level rather than targeting specific injury sites
Although their role in CIPN has been known for a long time, there are limited reports about the interaction between these factors and TRP channels in the literature
Neurological and Cognitive Applications GHK-Cu promotes nerve outgrowth with increased production of nerve growth factor (NGF), neurotrophin-3 (NT-3), and neurotrophin-4 (NT-4)
It has also been shown that immune system cells and DZN-induced immunotoxicity produce ACh through the non-neuroma lymphocytic cholinergic system [22]
This activation involves phosphorylation and nuclear accumulation of TAp63 in oocytes, which subsequently triggers the expression of pro-apoptotic Bcl-2 family members, including PUMA, NOXA, and Bax, initiating the apoptotic pathway (Suh et al., 2006
