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Research shows BPC-157 treatment accelerates healing of transected tendons through promotion of tendon outgrowth, cell survival, and cell migration via regulation of ERK1/2 phosphorylation and downstream targets including c-Fos, c-Jun, and Egr-1key molecules involved in cell growth, migration, and angiogenesis

Greene ponders, is there a potential risk for something similar to what you see with synthetic growth hormone

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The key question is not whether these controls exist, but whether they are producing the outcomes they were designed to achieve