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TIGIT is involved in mediating NK cell self-tolerance to maintain regenerative hyperplasia by impeding NK cell activation and decreasing IFN- production

Activating pathways for creation of NADPH has been shown to protect RPE cells from oxidative damage 74 , and our findings indicate that for developing mouse retinas with RP in which all rods have been lost a possible intervention strategy would be to stimulate synthesis of NADPH

To assess the ability of hepsin overexpression to protect against APAP-induced hepatotoxicity, a lethal dose of 600 mg/kg APAP was first administered to mice in each experimental group (Fig