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Heart failure (HF) is currently considered a systemic multi-organ syndrome fundamentally driven by metabolic failure, and carnitines, a crucial metabolite in fatty acid metabolism, transport long-chain fatty acids into myocardial mitochondria to produce energy as a source of ATP generation [1] (Fig

A comprehensive evaluation is necessary to determine the appropriateness of medication, along with regular follow-up and monitoring of blood carnitine levels and overall health status

and Rampello, L

These results should not be labeled as direct CPT1 activity unless the method was designed to isolate that step