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These findings suggest that DCA- or nutrient-induced GLP-1 secretion was inhibited by pasireotide via SSTRs-Gi-dependent inhibition of cAMP levels

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This case underscores the importance of recognizing how GLP-1 RA-induced motility changes can interact with pre-existing risk factors, highlighting the need for careful patient selection, close monitoring, and early intervention in high-risk individuals

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