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K., Cruzat, V

In this study, we found that in vitro exposure to IL-1 was sufficient to induce pT H 17 cells, driving an increase in CD38-expressing cells, whereas in vivo targeting CD38 consistently decreased cortical IL-1 in the AD brain, suggesting that CD38 treatment could lead to IL-1-mediated modulation of T H 17 cell pathogenicity

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Overall, discontinuation rates due to adverse events were low, with 5.9% for CagriSema versus 3.5% for placebo in REDEFINE 1 and 8.4% with CagriSema versus 3% with placebo in REDEFINE 2

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