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Previous studies demonstrate that sequence hybridisation of glucagon-like peptide-1 (GLP-1), glucose-dependent insulinotropic polypeptide (GIP) and glucagon (GCG) into a unimolecular triple agonist (GLP-1/GIP/GCG) effectively enhances glycaemic management relative to conventional mono-agonist treatments in diet-induced obese mice [1, 2]
Glutathione Reduced 500mg c thnh phn g
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These converge to induce serine phosphorylation of IRS-1, which inhibits its tyrosine phosphorylation and subsequent activation of the PI3K-Akt pathway, ultimately hindering GLUT4 translocation and glucose uptake (94, 95)
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