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Researchers exploring these combinations should proceed cautiously

doi: 10.1002/cam4.601 399

in vitro experiments showed that SGK1 knockdown alleviated erastin-induced downregulation of Slc7a11, GPX4, GSH, and GSSG, while reducing lipid peroxidation, iron accumulation, and mitochondrial damage upregulation, suggesting that SGK1 modulates ferroptosis processes through inflammatory pathways in CHD (44)

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Because of this dual role, MOTS-c is often described as a mitokine a mitochondrial-derived signaling molecule that influences cells throughout the body

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