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These pathways likely contribute to the neuroinflammatory state underlying neuropathic pain

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For example, a recent study indicated that unlike in mouse islets where tirzepatide stimulates insulin secretion predominantly through the GLP-1R, the therapeutic effects of tirzepatide on human beta and alpha cells require engagement of both the GLP-1R and the GIPR, and in addition, tirzepatide increased glucagon secretion in isolated islets [207]

doi:10.1016/B978-0-12-384964-9.00011-6

Role of bile acids and GLP-1 in mediating the metabolic improvements of bariatric surgery