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Immune checkpoint molecules, tumor-associated macrophages, regulatory T cells, and myeloid-derived suppressor cells can attenuate the efficacy of ferroptosis and cuproptosis inducers, thereby forming a drug-tolerant microenvironment that promotes therapeutic resistance and early recurrence
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doi:10.1016/j.phrs.2018.08.019
Pre-treatment scores that measure the severity of AAH, such as the MDF, MELD, age, bilirubin, international normalized ratio, creatinine (ABIC score), and the Glasgow alcoholic hepatitis score, all reliably forecast short-term mortality [4,8-11]
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