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Originally designed to manage type 2 diabetes, GLP-1 receptor agonists mimic the effects of glucagon-like peptide-1 , a hormone that regulates appetite, blood sugar, and fat metabolism

Among the most well-understood behavioral mechanisms of GLP-1 in the brain is its ability to restrict feeding behavior through its targeting of the PVH of the hypothalamus and the central amygdala [50, 54]

This combination can increase the risk of falls, disability, and metabolic disease even in those with high body weight

Spending time eating instead of rushing gives your gut a chance to send satiety signals properly

Given that, it makes sense that GLP-1 drugs might help