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Ozempic and other semaglutide drugs are designed to mimic GLP-1 by sending signals to GLP-1 receptors in the body, increasing insulin production, inhibiting glucagon secretion, slowing the time it takes your body to digest and process food, and artificially tricking your brain into telling your body that its full even though youre technically in a caloric deficit

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Similar caution extends to patients on potassium-sparing diuretics, aldosterone antagonists, or angiotensin-receptor blockers, all of which blunt renal potassium excretion