The stress + UFe also enhanced GSLs content, though to a lesser extent than stress + FFA, indicating partial activation of these defense-related secondary metabolites
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GST isoforms (GSTM1, GSTP1, GSTT1) are expressed in the cytosol, mitochondria, and nucleus across multiple organs, including the liver, kidneys, lungs, brain, and placenta, exhibiting broad substrate specificity against electrophiles (molecules deficient in electron pairs) and ROS (2733)
The malnourished state of the patient could be a contributing factor to low glutathione stores, leading to high 5-oxoprline levels [13]
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