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However, after 8 weeks of RT there were no significant differences in any of the measured variables

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Sensorgrams were analyzed using a global fit to the 1:1 Langmuir binding model

Thus, CHAC1s role in inducing ER stress and ferroptosis highlights its potential as a therapeutic target to increase the efficacy of DTX in treating castration-resistant prostate cancer ( In vivo experiments confirmed that knocking down miR-432-5p in CAFs enhanced CHAC1 expression, increased ferroptosis, and improved the efficacy of docetaxel in reducing tumor growth

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