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It provides broader context for Dihexa's mechanism of action and its position within the angiotensin IV receptor research field, evaluating both the strength and limitations of the existing preclinical evidence base

2020;8DOI

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Rinne et al., 1977) and probenecid-induced accumulations of HVA in CSF (Rinne et al., 1975, 1977), indicating that the drugs reduced the turnover of endogenous dopamine