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We reasoned that overlap of this second model with DA release supports a conclusion that links DA release to lack of rewards at reversal with the surprise component of RPEs more generally rather than to a change in valence

Other investigations relate bacterial LPS, altered bile acids, and tryptophan-derived metabolites to microglial priming, BBB malfunction, and worsening of -synuclein aggregation, giving multi-modal evidence of microbiota-driven neurotoxicity [55]