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In vitiligo, there is a Treg dysfunction, although it is not known exactly whether due to the inability to migrate to the skin, decreased numbers, or activity suppression.91 In animal models of vitiligo, there is control of disease progression and repigmentation of lesions with the re-establishment of the Treg population.92 Alterations of the adaptive immune system are demonstrated in both segmental and nonsegmental vitiligo, despite different reactivity patterns

These mechanisms make GLP-1 crucial for both glucose control and appetite regulation, making it a key target for therapeutic interventions in diabetes and obesity

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