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Lowering of the mitochondrial acetyl-CoA:CoA ratio would then favour glucose oxidation (Fig

Interestingly, Increasing the bio-availability of N-3 PUFAs [14], but not enriching the diet with a combination of micronutrients [15] early in life protected against the ES-induced reduction in neuronal survival

1.11.1.7.) and catalase (CAT, E.C

This results in derepression of the operon, allowing cellular accumulation of SQR, CstA, CstB, and a candidate TauE 23-25