Acute exposure induces a transient pro-oxidant state, while repeated treatments are generally well tolerated, likely due to hormetic adaptation ( Optimization of HBOT treatment protocols, including pressure levels, exposure duration, and session frequency, represents a practical strategy for minimizing oxidative injury while preserving therapeutic efficacy ( In experimental models characterized by high oxidative burden, such as systemic inflammation, toxic injury, and ischemia ( 6 Conclusion Overall, the analyzed literature suggests that the interaction between NAC and HBOT is neither universally synergistic nor antagonistic, but rather depends on the balance between pathological and signaling oxidative stress
Tumori 73 , 2528 (1987)
Kaul K, Apostolopoulou M, Roden M
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