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found that combination therapy was associated with a greater reduction in HbA1c of -0.74% compared to SGLT2i alone, while Chen Li et al

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TNF- damages cardiomyocyte mitochondria by inhibiting glycolysis and reducing mitochondrial adenosine triphosphate (ATP) production through promoting oxidative and nitrosative stress, which can result in compromised cardiac systolic function and unfavorable cardiac remodeling [83, 84]

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