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GSH imbalance and depletion are reported in many other brain disorders, including autism [28,29], bipolar disorder [30], schizophrenia [31], amyotrophic lateral sclerosis [32], and multiple sclerosis [33]

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The hyperglycemia-related increased proximal tubular sodium reabsorption results in lower sodium levels at the level of the macula densa increasing GFR via inhibiting TGF, further augmenting the amount of filtered and reabsorbed sodium with increased ATP demands

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