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Conversely, the ER stress inhibitor (4-PBA) did not mitigate PFOS-phosphorylated ERK1/2 levels at 1 h of PFOS treatment in RTCs, but it reduced the level of LC3BII (an autophagy biomarker) at 24 h of PFOS treatment in RTCs (Fig

Understanding these mechanisms provides key insights into the pathology of cadmium-induced hepatotoxicity (158)

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To determine whether increases in intracellular GSH directly enhance expression of T2-signature proteins in HAECs, we lowered intracellular GSH levels by inhibiting SLC7A11 using the chemical inhibitor erastin (16)