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A similar scenario has in fact been reported recently in an alcoholic liver disease model in which long-term alcohol consumption activated NF-B in Kupffer cells, whereas NF-B activation in hepatocytes was inhibited 45

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It remains unclear to which extent the protection mediated by HGF-MET from the excitotoxic insult is mechanistically linked to the protection from other forms of regulated cell death, such as apoptosis and parthanatos

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