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However, when it was reported that GLP-1 was a substrate for DPP-4 in pharmacological in vitro kinetic studies (18), this was quickly followed by the demonstration that the metabolite generated by DPP-4 cleavage was the major circulating component of GLP-1-like immunoreactivity in healthy individuals (19) and that the same metabolite formed rapidly following exogenous administration of GLP-1 in both healthy subjects and those with T2DM (20)

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The primary finding is an increase in sinus rhythm (a faster normal heartbeat)

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Also, oral dose of encapsulated glutamine did not stimulate consistent increase in GLP-1 and insulin secretion in type 2 diabetes patients [34]