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SEMA, as a treatment for obesity, has shown nephroprotective effects, with a 22% reduction in the composite renal endpoint in the SELECT33 trial in patients without diabetes, supporting the role of SEMA in the management of the cardio-renal-metabolic syndrome

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Inhibiting DPP4 in a mouse model of HHT1 results in a shift towards regenerative macrophages and reduces fibrosis after myocardial infarction

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