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Description

Studies into the molecular mechanisms underlying the time of day differences post ischemia revealed that ischemic/reperfusion injury at ZT 18 had reduced infarct volume, edema, neurological deficits, apoptotic death, and improved neuronal survival compared to other time points (ZT0, ZT6, ZT12, and ZT18), coinciding with a decrease in PRAS40 and increased expression of Bmal1, Per1, Clock, AKT, Erk-1/2, mTOR, S6 , and BAD (Beker et al., 2018)