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It is known however that lipid nanodomain-specific cAMP generation, measured using the raft-anchored cAMP sensor T Epac VV -Lyn (112), is selectively enhanced by the -arrestin-biased GLP-1RA exendin-asp3 over the G protein-biased exendin-phe1, matching the relative propensity of these agonists to stimulate segregation of GLP-1R into lipid rafts (71)

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Moreover, the brain regulates Hcy levels by exporting them to the external circulation [69]