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The extent of systemic inflammation and the magnitude of increase in circulating levels of GLP-1 correlates with outcomes in people with sepsis or myocardial infarction [130, 131], although levels of GLP-1 are not universally elevated in people with acute or chronic inflammation [132]
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In the pancreas, these agents enhance glucose-dependent insulin secretion while suppressing inappropriate glucagon release, improving glycemic control with a low risk of hypoglycemia when used alone (though this risk increases when combined with insulin or sulfonylureas)
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