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These quality parameters are especially relevant in studies where inflammatory signaling or immune interference must be carefully controlled

Who May Be a Candidate for GLP-3 Therapy

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Recurrent associations between dysbiotic microbial patterns and poor metabolic response further support the hypothesis that gut microbiota may contribute to interindividual differences in response to incretin-based therapies, although definitive mechanistic evidence in humans remains limited (136), and causality between specific microbial taxa, functional pathways, and GLP-1 RA responsiveness cannot be inferred