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Most recently, AAV9-mediated SLC52A2 gene replacement normalized FMN/FAD levels, rescued spinal motor neurons and reversed motor deficits when delivered pre-symptomatically, underscoring the feasibility of curative therapy (19)

Redox-based regulation of apoptosis: S-glutathionylation as a regulatory mechanism to control cell death

This distinction places it in a new category of metabolic treatment

A 2025 comprehensive review in Medical Science Monitor covering studies from 20162025 confirmed that GHK-Cu-based formulations enhance fibroblast migration, ECM remodelling, collagen and elastin synthesis, and wound closure across multiple model types

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