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It has been shown that GLP-1RA semaglutide can improve working and spatial memory in transgenic AD modelling APP/PS1/tau (3xTg) mice, promote the release of anti-inflammatory factors and inhibit the production of pro-inflammatory factors in the cerebral cortex and hippocampus, as well as reduce the deposition of -amyloid in the CA1 subfield of the hippocampus

In vitro (cAMP production functional assay), hybrid 24 was found to be equally potent to semaglutide (Supplementary Fig

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The abstract (but not the complete study) for the SURMOUNT-OSA trial was just published in the New England Journal of Medicine