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However, early attempts to apply rotenone as a PD model found that high doses of rotenone induce widespread brain damage rather than specifically targeting the nigrostriatal system, which might interfere with the value of relevant studies (Heikkila et al., 1985

observed that the activation of NLRP3 inflammasome and NLRP3 expression were weakened by cGAS inhibitor while STING agonist reversed this inhibition

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