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Liraglutide, in particular, shares a 97% amino acid sequence with the native human GLP-1, contributing to its therapeutic effectiveness [58]

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It occurs naturally in human plasma, saliva and urine

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Therefore, localized immunometabolic disturbances, particularly defective butyrate metabolism and dysregulated tryptophan catabolism, disrupt the Th17/Treg balance and compromise epithelial integrity in IBD, underscoring the gut microbiome and microbial metabolites as actionable therapeutic targets