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TMAO is formed in the liver from trimethylamine (TMA), a substance produced by gut bacteria from food-based compounds such as choline, carnitine, lecithin, betaine, gamma-butyrobetaine, and ergothioneine

Additionally, TGF-1 promotes fibrosis by enhancing autophagic flux through increasing cellular autophagosomes, thus reducing the level of autophagy in fibrotic tissues, which may be a potential target for anti-fibrotic therapy (Weiskirchen and Tacke, 2019

Similarly, another smaller study, BEDIP, with a shorter duration, found improvements in a combined neuropathic symptom and sign score after 3 weeks of treatment with 400 mg of benfotiamine daily [72]

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