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The mouse model used for these experiments is a general knockout and we can therefore discount the possibility that GLP-1 exerts its inhibitory action by paracrine effects secondary to activation of GLP-1Rs in delta and beta cells, suggesting that GLP-1s glucagonostatic effect involves a receptor distinct from GLP-1R

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Instead, weight-loss treatment plans are customized to the individual and built around what is medically appropriate, evidence-based, and currently legally available

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The findings are preliminary and observational, not proof of causality