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5C) revealed that mTOR inhibition slightly impaired responsiveness to glucose, but entirely abrogated cellular ability to increase ECAR to compensate for oligomycin-induced blockade of mitochondrial ATP production
Loss of Angiotensin-Converting Enzyme-2 Exacerbates Diabetic Cardiovascular Complications and Leads to Systolic and Vascular Dysfunction: A Critical Role of the Angiotensin II/AT1 Receptor Axis
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