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Beyond the observed increase in female lifespan, we show that pharmacologic blockade of IGF-1R signaling preserves, to various degrees, many aspects of healthspan, including neuromuscular function and endurance, and attenuates the age-related increase in diastolic dysfunction, systemic inflammation, and neoplasias in females

Similarly, oxidative stress and cell death pathways interact with each other in complex ways that complicate therapeutic targeting 10

Experiments have shown that the specific inhibitor of PIEZO1 can significantly reduce iron overload, inhibit mitochondrial oxidative stress, and block the characteristic changes of ferroptosis induced by mechanical stress (Liu et al., 2025)

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