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Mitochondrial DAMPs activate PRRs on glial cell surfaces, leading to the release of pro-inflammatory factors, activation of inflammatory pathways, and exacerbation of mitochondrial damagecreating a vicious cycle of mitochondrial dysfunction and neuroinflammation [276, 331]

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the subsequent poly ADP-ribosylations of a number of signaling and structural molecules by PARPs were shown to facilitate DNA repair at DNA strand breaks (44)

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