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For the short-term MIA model, 120 animals were divided into seven groups: saline/vehicle group (n = 19 mice), MIA/vehicle group (n = 18), all MIA/liraglutide groups (n = 17), and dexamethasone group (n = 15)

This provides high-fidelity information on diseases and treatments in the whole period in which GLP-1 RAs and DPP-4is have been marketed in Denmark, allowing an unbiased study population with very little missing data, and providing results which are highly generalizable to other similar populations

Previous studies have demonstrated that gut-derived GLP-1 activates vagal sensory nerves, triggering insulin secretion via brain-autonomic nervous system pathways, a mechanism referred to as the "neuro-incretin effect" [51, 59]

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By calming exaggerated reward signals, GLP-1 drugs may reduce the pull of alcohol, nicotine, sugar, and possibly other substances