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Using nanoBRET technology, we focused on ligand-stimulated receptor recruitment of G i and -arrestin2, initiators of G protein-dependent and G protein-independent signaling pathways, respectively 56

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Similar changes in the expression of the above markers of ER stress, oxidative stress, EMT and ECM accumulation were also observed in HK-2 cells stimulated with TGF-1, accompanied by ER and mitochondrial damage, as shown by TEM

The results are noticeable after a few weeks