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The chief treatment options for UC are aminosalicylates, calcineurin inhibitors, immunomodulators, Janus kinase (JAK) inhibitors and corticosteroids (Fig
Amphetamines disrupt VMAT2 function and vesicular uptake through a combination of mechanisms: The Weak Base Effect (pH Gradient Collapse) : Amphetamines are lipophilic weak bases (pKa of 9.9) that can readily diffuse across both the plasma and vesicular membranes to accumulate inside synaptic vesicles (Wimalasena 2011)
This can minimise side effects and give your body more time to get used to the medication
Preparation of synthetic carrier: Synthetic carrier is the material used to carry amino acid and react in solid phase synthesis
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