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Although GCs are important in maintaining lipid homeostasis, an excess of GCs can lead to an increase in circulating free fatty acids and cause lipid accumulation in skeletal muscle and liver also associated with insulin resistance [80]

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However, it is now known that full potentiation of GHRH in-vivo is due to GHS suppression of somatostatin as well as to stimulation of GHRH releasing neurons in the arcuate nucleus 6