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For example, Nrf2, a representative antioxidant gene, activates the transcription of ferritin heavy chain 1 (a protein involved in iron storage) and heme oxygenase-1 (a protein involved in heme breakdown) to reduce labile iron and regulate iron metabolism and oxidative stress [15,45]

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introduces a metabolic confounder that is not present in the other trials, which may affect both the response to GLP-1 agonists and the severity of OSA at baseline and follow-up [7]