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That is part of the process

doi: 10.1210/jc.2016-2587 64

Within the brain, insulin resistance triggers the dysregulation of sphingolipid metabolism, leading to the accumulation of neurotoxic ceramides and a deficit in neuroprotective sphingosine-1-phosphate (S1P) ( Therefore, we hypothesized that the therapeutic action of SE involves a significant gut microbiota remodeling, and that this remodeling is associated with key metabolic shifts in the gut-brain axis, including bile acid and central sphingolipid pathways

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The time course of action of insulin and insulin analogs such as NOVOLOG may vary considerably in different individuals or within the same individual