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These properties raise the possibility that PT150 could modify stress-dependent drivers of weight regain and homeostatic setpoint defense, particularly when used in combination with agents that induce robust initial weight loss, such as GLP1R agonists

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doi: 10.15252/embr.202256614 Summary Keywords mitochondria, infection, mitochondrial dynamics, pathogens, host defense Citation Yang Y, Zeng Y, Liu Z and Huang J (2025) Pathogen-induced mitochondrial dysfunction: mechanistic insights, immune crosstalk, and therapeutic opportunities