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The gut releases an incretin hormone in response to food intake, particularly when carbohydrates and fats enter the small intestine

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The data presented here demonstrate that the potency of GcgR agonism defines the maximal weigh-lowering capacity of the triple agonist, presumably through multiple mechanisms of increased energy expenditure, insofar as increasing in vitro GcgR potency with respect to cAMP production prognosticates in vivo weight reduction

doi: 10.3390/molecules29163941 255